ResveratrolConsumer
avert online
consumer fraud
ResveratrolQuiz
test your knowledge
New E-Book
How the world got lost on
the road to an anti-aging pill
Subscribe to our newsletter to receive email notifications when new articles are posted.
June 19, 2020: by Bill Sardi
Of acute concern are “immunity passports” that are a proposed requirement for air travel. Those individuals who developed immunity without antibodies would not be granted passports.
Ben Lazarus, a features editor for the UK’s MAIL ON SUNDAY, tested positive for COVID-19 in March of 2020 but subsequently tested negative in three successive tests in the following month. Does this mean natural immunity is not lasting and even vaccination would wane and repeated immunizations will be required. No one knows for sure.
A just-released study of hospital workers who were exposed to infected patients reveals only 4% of 23,000 had antibodies, but researchers estimate 25% contracted the COVID-19 disease.
Professor Karol Sikora, an advisor to The World Health Organization says: “the majority of (COVID-19 coronavirus) infected patients have a negative antibody test even though they have had the coronavirus.” Based upon available data, maybe only 10% of COVID-19-infected patients develop antibodies, says Dr. Sikora.
Maybe potentially-toxic heavy metals like aluminum or mercury (Thimerosal) would be needed to provoke a stronger antibody reaction.
People infected with the SARS coronavirus a decade ago generated protective immunity for up to 10 years. So COVID-19 may be a worse monster than first realized because immunity doesn’t appear to be long-lasting.
Dr. Arthur Edridge of Amsterdam University, has tracked ten coronavirus-infected individuals from 1985 to 2020. These patients experienced bouts of infection between 3 and 22 times each over a 35-year period. Dr. Edridge concludes the human body only develops short-term immunity from coronaviruses. In another recent study, antibody levels declined by 50% in 6-months and 75% after nine months.
Anthony Fauci, director of the US National Institute of Infectious Diseases, says natural immunity (without vaccines) for coronaviruses is not long-lived, inside of a year.
Vaccinologist Greg Polland MD of the Mayo Clinic says immunity for seasonal coronaviruses may last several years or as little as 80 days.
It’s potentially a bonanza for vaccine makers. A COVID-19 coronavirus vaccine may have to be administered over and over.
That doesn’t rule out there will be other ways to produce long-term antibodies, but it certainly casts a pall over current efforts to eradicate this pathogenic cold virus.
Human populations would be perpetually held in lockdown if antibodies are the only measure of effectiveness of natural immunity or vaccine induced immunity. Few will be able to obtain an “immunity passport.”
Only months into the viral pandemic researchers are grappling with a different phenomenon than they have faced with any other virus (save for Dengue Fever) — antibody-enhancement. The natural antibodies that are produced to quell COVID-19 infection, infect immune cells such as macrophages that normally ingest virally-infected cells. The immune system itself is infected.
COVID-19 coronavirus deactivates interferon, a natural anti-inflammatory agent. Interferon is the “initial alarm” of the immune system. COVID-19 turns off the fire alarm.
Dr. Karol Sikora points to T-cells which are activated prior to antibodies as being the likely agent that overcame COVID-19 infection.
Gus Dalgleish, professor of oncology at the University College London, says he believes “the majority of people fight the virus with T-cells, based on research so far.” T-cells decline with advancing age notes Dalgleish.
A study conducted by Rockefeller University examined blood samples of 149 recovered patients and found every patient’s immune system generated antibodies, just not enough of them, says a report in the Daily Mail (June 7, 2020). Maybe antibodies simply aren’t permanent. So-called memory T-cells produce antibodies that may last a lifetime. Maybe that sub-set of T-cells isn’t being produced sufficiently.
T-cell activity is not a current measure of vaccine effectiveness. Only antibodies are. And therein lies the problem. If you don’t develop measurable antibodies you are going to get thrown back into quarantine or refused a “immunity passport” for air travel. The scientific community knows it is on a road to nowhere and isn’t responding in a timely manner.
There are two types of T-cells: CD4s for immune maintenance and CD8 for eradication of pathogens. When these CD4 and CD8 T-cells become overactive, an autoimmune reaction takes place. The body attacks itself and destroys tissues and organs. Various drugs are employed to dampen CD4 autoimmunity/allergy, but these drugs also impair immunity and therefore, vulnerability to infectious disease.
CD4 T-cells have distinct advantages over antigens (allergens, germs). The subcutaneous (under the skin) administration of antigens (i.e. vaccination) allows a lower dose of the immune provoking CD4 cells to be activated. The objective is to normalize the immune response so as not to damage tissues and organs, what is commonly experienced in autoimmune disorders.
While modern medicine is in denial, there is a growing body of scientific literature showing a connection between vaccination and autoimmunity. The human immune system is designed to recognize the difference between “self” and “non-self” molecules. “Non-self” molecules like bacteria and viruses are attacked and abolished by an army of white blood cells and T-cells. When the immune system is overly sensitive it can be switched on by “self-molecules,” your own tissues and organs, not just “non-self” proteins. Vaccines are documented to cause confusion between “self and non-self-molecules” to induce chronic autoimmune reactions.
Mayo Clinic’s Greg Polland says: “there’s an immunological secret locked up in the long class of coronaviruses that we don’t understand yet.” (Read below)
In 2004 researchers at Harvard reported a red wine molecule (resveratrol), in a superior manner to other molecules, activates a survival gene called Sirtuin1. Any physical or mental stress activates Sirtuin1.
When studying the immune response, the removal of the Sirtuin1 gene from living cells overly sensitizes the ability of the body to tolerate and withstand infectious or allergic agents. In other words, the Sirtuin1 gene keeps a lid on the human immune system.
In an oversimplified explanation, T-cells can become excessively active and this becomes autoimmune disease. Resveratrol does the opposite via the Sirtuin1 gene.
With the Sirtuin1 gene intact, some level of allergens and germs are “tolerated.” Resveratrol via activation of Sirtuin1 inhibits T-cell activation and reduces inflammation. Therefore, resveratrol is an immune system normalizer.
Resveratrol calms a cytokine (sî-toe-keen) inflammatory storm, the same storm that is involved in acute respiratory attack posed by COVID-19 coronavirus. Resveratrol has been demonstrated to inhibit infection specifically by coronaviruses.
In the laboratory animals were immunized with a peptide (two or more amino acids) to provoke autoimmune inflammation in the brain (encephalitis). Resveratrol was then employed to activate the Sirtuin1 gene and quell the autoimmune riot.
The Sirtuin1 gene is very abundant in the thymus gland where T-cells are produced. The chart below shows how well resveratrol (green) inhibits autoimmune encephalitis in animals after vaccination.
This knowledge has application for rheumatoid arthritis, lupus, multiple sclerosis, diabetes, and many other autoimmune disorders, but goes ignored in modern medicine.
Posted in Resveratrol
Add comments »